Bored Druid ThreadFollow

Talking about coffee, I've actually managed to cut down on my coffee usage.
I'm down to two cups a day now.
Very huge cups.

I'll give Chicago as a Top 10 City, but Seattle? That's like the birthplace of the coffee suckin' hipster. Let it sink into the ocean for all I care for inventing that plague.

I have to just pop in and do my +1 ever couple hundred replies or so... It's only right.

So much talk about eating protein. Not sure if excited or just scared...

Thoroughly aroused?

Did they ever follow up on that to your knowledge? Or should I dive back into pubmed?

L-tyrosine, an essential precursor for dopamine
and norepinephrine, has been shown to be beneficial
for attention in adults with ADHD.81 Around
5-10 percent of ADHD cases respond to L-tyrosine
supplementation and are most likely those individuals
who have impairments in neurotransmitter
metabolism.82 In one case study evaluating the
effect of tyrosine supplements on a patient with
ADHD and co-morbid phenylketonuria, tyrosine
was found to reduce ADHD symptoms, possibly
through the action of augmenting dopaminergic
activity.83

Our first finding was that L-tyrosine was therapeutically
effective after 2 or more weeks. This delay is
similar to that found by Gelenberg et al. (9) in the
tyrosine treatment of depression and to that encountered
with the use of tricyclic antidepressants and
monoamine oxidase inhibitors in the treatment of
depression. The delay seen with antidepressants parallels
changes in receptor function and suggests that
analogous changes may have to occur before L-tyrosine
becomes therapeutically effective. In our previous
trials of d,l-phenylalanine (8), the pharmacological
response was immediate, so we did not predict the
delay in L-tyrosine response; therefore, a placebo response
is not likely, even in an open trial.
The second finding was that all the patients who
responded to L-tyrosine became tolerant to its therapeutic
effects. The clinical reports on the effects of
L-tyrosine in depression (9, 10) do not discuss the
length of time that the treatment remained effective or
whether tolerance developed.
In conclusion, the three precursor studies-L-dopa
(5), d,l-phenylalanine (8), and L-tyrosine-have not
elucidated the putative functional defect in attention
deficit disorder, residual type. Furthermore, none of
these substances appears to have any clinical utility in
the treatment of the disorder.